The Technocrats’ Virus
The technocrats needed a virus for the Great Reset and SARS-CoV-2 was the perfect design. Being just a little bit deadly, this virus helped establish a calm and orderly pandemic for which they already had the ‘cure’; the coronavirus vaccine design that was patented in 2017.[i] The technocrats’ virus is a new kind of bioweapon that works on many levels but the ultimate goal is total control by way of the global ID.
Virus – Vaccine – Global ID
Right from the start, the Reset Mafia have worn their hearts on their sleeves, and made it clear they want everyone to get the jabs and the stupid ID and shut the f**k up about 5G. These are important clues about the world they intend to create – they know that most people have a smartphone already, so all they have to do is link it to the ID and they’ll be able to introduce Central Bank Digital Currencies and a Universal Basic Income. The ID has to be linked to a smartphone because it’s got a battery and read/write capabilities. This means it can be used to manage the digital credits that’ll probably be doled out for good behaviour, and it also allows privileges to be revoked! Each person will become a node in the Internet of Things, linked and controlled by 5G and the centralized blockchain.
Making the Virus for the Vaccine
If you want to make a coronavirus vaccine, you’re gonna need an actual coronavirus; and scientists such as Ralph Baric and Zeng Li Shi have proved it’s possible to create specific types. Some of the most famous coronaviruses were created at the Wuhan Institute of Virology (WIV) using gain-of-function research, but that doesn’t mean they were all dangerous. In fact, researchers at WIV usually found that when they genetically engineered bat-coronaviruses so they could infect humans, the viruses often became LESS DANGEROUS. Gaining one function led to the loss of another, e.g. when they were able to grow well in human cell cultures, they caused less damage to those cells. They would be able to infect humans without being highly pathogenic, and this is how SARS-CoV-2 seems to be. The work done at WIV was heavily funded by the NIH and DARPA, and some of the viruses they found/created have been used in coronavirus vaccine patents.
A lot of experts think the SARS-CoV-2 virus could have been man-made, especially the spike part, and it’s the spike part that’s in the vaccines! Several unusual features about the spike have aroused suspicions, but what really takes the biscuit is the fact that it’s somehow managed to acquire a furin cleavage site. This design makes it a perfect match for the 2017 Prefusion coronavirus spike proteins vaccine the NIH was working on with Moderna in the years preceding 2020. And although the first sequences of SARS-CoV-2 were submitted by China, it’s the NIH who annotated the sequences of the genome referred to as Wuhan-Hu-1 or MN908947.3 and this is the version that’s in the vax.
Where viruses come from
The origin and effect of viruses is a matter of some debate[ii], mainly because it’s impossible to study them in their natural environment, i.e. in the body. Viruses are little blobs of genetic material that come out of our cells, and some people think they’re actually a symptom of disease rather than the cause of it. However, this is an argument that relates to natural diseases and how viruses came to exist in the first place[iii], but this argument is almost irrelevant when it comes to man-made viruses because we know how they came to exist.
Whatever the causes of naturally-occurring diseases may be, it’s definitely possible to manufacture little blobs called viruses; it’s also possible for people to transmit them to others, and this can sometimes cause disease. Viruses and other ‘cell stuff’ have always been grown artificially, so essentially they are always ‘man-made’. The methods for growing them have changed a bit, but viruses have ALWAYS been grown in cells of some kind, so SARS-CoV-2 is nothing new in this regard. For the first two hundred years, inoculation was performed using pus or blood from sick people and animals; after that, chicken eggs were used, and in more recent years, viruses have been grown in cell cultures. This is a bunch of cells that are kept alive by feeding them a broth made from fetal bovine serum (blood from aborted baby cows), antibiotics and some nutrients. This is a widespread practice in the genetics industry because it’s the best they’ve got, but it’s clearly deficient as a model for how viruses behave in the human body.
If you wanted to avoid causing mayhem, and to safeguard your own health, it would make more sense to create the illusion of a pandemic by using a virus that’s only a little bit deadly. Still, it’s no surprise that people started to doubt the existence of SARS-CoV-2 quite simply because there’s been no evidence of a raging pandemic. This led to people demanding to see proof of an isolated SARS-CoV-2 virus, when in fact the term ‘isolate’ is used by scientists to refer to a cultured organism. For example, the patent for the Canadian SARS-CoV reference genome (Tor2) describes it like this:
“An isolated microorganism (such as a virus, bacterium, fungus, or protozoan) has been Substantially separated or purified away from microorganisms of different types, strains, or species. Microorganisms can be isolated by a variety of techniques, including serial dilution and culturing.”
So there’s not much point in petitioning for an isolate of SARS-CoV-2 to prove its existence because it seems such proof has never been provided! For any virus! Apparently, viruses can’t be isolated because there aren’t enough in samples taken from people, and because they die if they’re not in living cells. They can be purified[iv] to remove unwanted genetic material (e.g. by using ultracentrifugation) but usually they’re just added to a cell culture to clone them. Besides, millions of SARS-CoV-2 genomes have now been sequenced and compared, and all the experts are very sure it’s the same virus. The methods they use to sequence the genome of a virus appear to be much the same as the way other genomes are identified, including the human genome. Genetic sequencing is definitely a fishing expedition and it does often rely on previously identified sequences and computer algorithms, but that doesn’t mean that everything the experts say about viruses is wrong. Perhaps, then, the ronascam is only partly dependent on misuse of the PCR test, and maybe some of the people who test positive do actually have some SARS-CoV-2 viruses in them, but most of them are asymptomatic quite simply because it’s not a particularly dangerous virus.
In the end, though, the only way we’re going to understand recombinant viruses is by listening to what the experts say; and in fact, the only way to understand what’s (supposed to be) in the vaccines is by learning from the people who mess around with viruses in the lab. Their work can help us to understand what kind of construct SARS-CoV-2 actually is, and why the experts think it’s really dangerous when the statistics indicate a low to zero risk.
Diseases caused by man-made viruses
One of the ways that lab-cultured viruses have been shown to cause disease is when they’ve been used in vaccines. This is well known to have happened with polio and measles; the vaccines sometimes caused a similar disease to the one they were supposed to prevent. In other words, some people got ill from the vaccine-virus, rather than the original, wild-type virus, and this was established using genetic sequencing. It’s also possible to inject people with vaccines that contain bits of a virus and to have them test positive for that disease even though they’re not ill. This is known as ‘vaccine-induced seropositivity’ or VISP, and has happened with a number of genetic vaccines for HIV, and even recombinant protein vaccines for hepatitis. It also happened last year with a coronavirus vaccine made by Uniquest in Australia – it contained tiny fragments of the genetic code for HIV and people were testing positive for HIV so it was scrapped. VISP can last for years – a whole lifetime, even – which suggests the thing they injected can persist for years. So what about SARS-CoV-2? Why are so many people testing positive for the virus after being injected with the ‘vaccines’? People in South Africa (where there’s an issue with VISP from HIV vaccines) were assured that they didn’t need to worry about getting VISP from the SARS-CoV-2 vax because most tests are looking for antibodies against the N gene, rather than sequences from the spike gene (which is what’s in the vax).
Testing transmission of viruses
Scientists have performed countless experiments with many different viruses that involve testing transmission between animals. These experiments involve testing the two theoretical routes of transmission:
- direct contact – the animals are placed in cages together, where they will come into direct contact frequently
- respiratory droplets – the animals are placed in separate cages in the same room, so they are near to each other, but not touching.
To begin the experiments, they check and make sure all the animals are free of the virus, then they inoculate a few of them to see if the other animals end up catching it. They can establish chains of transmission, too, where the animals who’ve ‘caught’ the virus are then able to transmit the virus to other animals, and so on. It’s even possible to genetically barcode the viruses they’re using so they can easily be distinguished during the experiment.
One of the most famous experiments that studied transmission of viruses involved making a flu virus more deadly by passaging it through ferrets. Viruses that come from one particular species aren’t normally able to infect another species, but they can be trained to adapt to a new host by passaging it in the target species until it mutates enough to be happy to live there. Fouchier et al genetically engineered the H5N1 bird flu virus so that it could infect ferrets, then harvested the virus from an infected ferret and used it to infect another one. They repeated this several times until they’d done ten passages of the virus altogether, and by this point, the virus had managed to adapt to its new surroundings (the nose of a ferret). They also passaged a wild-type virus that didn’t have these mutations. To test for transmission through the air, the scientists inoculated four ferrets with the new mutant virus, and two with the wild-type virus that had also been passaged ten times. Then they put some uninfected ferrets in cages next to the infected ones but not close enough for direct contact. Ferrets exposed to the wild virus remained uninfected, but most of the others did get infected with the mutant, and were then able to transmit it to two other ferrets. The experiment also showed that the viruses made the animals ill:
“In the course of the airborne transmission experiments, the ferrets showed signs of illness, including lethargy, loss of appetite, and ruffled fur. One of the directly inoculated ferrets died, but all those infected via airborne viruses survived.”
Fouchier’s team then sequenced the viruses and found they had all developed the exact same mutations, and they used this new variant strain to inoculate some more ferrets at high doses, and after just three days, they were all either dead or close to death.
Deliberate Release of the Virus
If we suppose that SARS-CoV-2 was indeed man-made, and is also somewhat contagious, it could have been deliberately transmitted in one or more locations. This could have been achieved in a number of ways; it could have been spread around at the Wuhan Games, added to injections in a hospital in Lombardy, or perhaps even applied to PCR swabs in New York. We will probably never know, but there’s no particular reason to assume the release began in Wuhan.
It’s perhaps possible that the first SARS virus was (also) deliberately released, back in 2002. The WHO declared that the outbreak was over in 2003, so the virus was said to have been eradicated, but was it?[v] And what about the other outbreaks? There was swine flu, bird flu, MERS, Zika and Ebola, all of which are viruses with a furin cleavage site like SARS-CoV-2. Were any of those engineered? Certainly, the MERS coronavirus outbreak helped further research into the spike protein, and in the years running up to the ronascam, several genetic MERS vaccines were patented and trialled, which is one of the reasons the SARS-CoV-2 vax were developed so quickly. MERS also kept the coronavirus hype alive, so that when a novel coronavirus was announced in 2020, all experts were quick to assume it was deadly.
Many questions to ask
- Why do the experts say that SARS-CoV-2 is dangerous? Was the first SARS-CoV really that deadly?
- Why does the rona seem to be man-made? And what on earth is a furin cleavage site?
- What kinds of coronaviruses were created by Baric, Shi and Daszak? How did this help the development of vaccines? What parts were played by the Vaccine Research Center, Scripps and Dartmouth College?
- What about the sudden discovery of a bat coronavirus called RaTG13 by Zeng Li Shi? Why was the Shanghai lab closed for rectification the day after they released the first version of the Wuhan-Hu-1 genome?
These questions will all be addressed in this upcoming series of articles.
It’s all about balance
Whether or not a virus or toxin causes disease is known to depend on a wide range of factors, such as stress, pollution, sickness, and diet. It also depends on how many viruses there are! But no matter how it got there, its fate within a body depends entirely on the conditions it encounters. Health is always a question of balance, and a major part of that balance is the health of our microbiomes. These are the little worlds inside of us that are teeming with microbes, some of which help our immunity and even our happiness! (About 75% of our serotonin is manufactured by these little bio-blobs!) They’re all trying to live in harmony with each other so they can survive, but vaccines can upset that balance, even when they don’t contain (the code for) a whole virus. It might also be what led to the so-called Spanish Flu pandemic because this was a time of widespread experimentation using vaccines that were mostly based on bacteria. Being clean seems to be what reduced viral infections the most.
Dismissing the existence of viruses might mean we ignore the role of our microbiomes and how the balance of microbes can be affected by genetic vaccines, such as the recent rise in cases of shingles.[vi] It might be more helpful to suggest positive actions to remedy this such as use of probiotics, e.g. Lactobacillus rhamnosus may inhibit the activity of the herpes simplex virus that’s said to cause cold sores.
If SARS-CoV-2 is actually real, it might mean there’s a risk of Vaccine-Associated Disease Enhancement (VADE) with the coronavirus vaccines, which could explain the rise in cases/adverse events. VADE has been associated with a number of vaccines in the past and antibody dependent enhancement is one of the problems that can occur.[vii]
And if there’s a chance that SARS-CoV-2 was released deliberately, it deserves a much closer look.
[i] Prefusion coronavirus spike proteins and their use (US20200061185A1; assigned to the NIH/DHHS/US Government in 2019)
[ii] The debate about viruses is hugely complicated because there are just so many of them (“~320,000 types of viruses are estimated to infect mammals”) and because it can be easy to confuse symptoms with other diseases, such as with covid. However, some have very distinctive symptoms which seem to provide better evidence. A good example is a syndrome that affects snakes; they tie themselves up in knots and begin stargazing. This was a mystery disease for a long time, but Joseph deRisi claims to have discovered a virus that causes it, as explained in this video. He begins by asking, “How might you find a pathogen if you don’t know exactly what you are looking for?”, then goes on to describe how and why he thinks he’s found the cause of the disease.
[iii] There’s no way to be sure which came first (the chicken or the egg?). It’s a bit like this with depression – people who are depressed are often told it’s because they’re not making enough of a neurotransmitter called serotonin, and yet people who are grieving also make less serotonin. All we can really say is that a reduction in serotonin correlates with being unhappy, in the same way that the appearance of viruses can correlate with the appearance of a distinct type of disease. And if using drugs to increase serotonin levels can create a sense of happiness, then maybe diseased cells from a sick person can induce the same sickness in somebody else. Would you call it a virus if it did?
[iv] The Tor2 patent defines ‘purified’ as being ‘more pure’: “The term ‘purified’ does not require absolute purity; rather, it is intended as a relative term. Thus, for example, a purified protein preparation is one in which the Subject protein is more pure than in its natural environment within a cell. Generally, a protein preparation is purified such that the protein represents at least 50% of the total protein content of the preparation.”
[v] Because the first SARS virus was said to be “eliminated”, scientists think it’s okay to use fragments of it for the PCR and antibody tests being used for SARS-CoV-2! The N gene of SARS-CoV is almost identical (97%) to the N gene of SARS-CoV-2 so there can be “significant cross-reactivity” when the N protein is used. There is also a degree of cross-reactivity with other coronaviruses, such as those that are said to cause the common cold. One study that tested antibodies in the blood of SARS survivors found they all had “significant levels of antibodies remaining in their blood 17 years after infection. Anti-N antibodies waned more than anti-RBD antibodies, and the latter is known to play a more important role in providing protective immunity.” (The RBD is part of the spike!) Also, “Anderson et al demonstrated in a cohort of 207 pre-pandemic samples that 5% reacted to the SARS-CoV-2 S proteins, 2% against the RBD, and 19% against N.”
[vi] It’s even happening to people who have not been jabbed! The chicken pox virus that can flare up as shingles is normally kept in check by TLRs, which are sensors in our immune system that recognise pathogens, and some of these sensors get deliberately hijacked by the alien genetics in the mrna vaccines (including TLR3 which also recognises the chicken pox virus!).
[vii] Also referred to as VAED (vaccine-associated disease enhancement), e.g. by the Brighton Collaboration who say adverse events in clinical trials should be monitored for potential cases. It’s interesting that they have higher standards for defining a case of VAED than the standards used to define covid! They state, “In the context of evaluating a case for VAED, it will be important to rule out other infections, comorbidities, drug effects, toxicities, etc. If no alternative explanation for the frequency or severity of illness is identified, including vaccine failure, VAED or VAERD may be considered.”